Vitamin B17 appears in abundance in untamed nature. Because B17 is bitter to the taste, in man’s attempt to improve tastes and flavors for his own pleasure, he has eliminated bitter substances like B17 by selection and cross-breeding. It can be stated as a general rule that many of the foods that have been domesticated still contain the vitamin B17 in that part not eaten by modem man, such as the seeds in apricots. Listed below is an evaluation of some of the more common foods. Keep in mind that these are averages only and that specimens vary widely depending on variety, locale, soil, and climate.
Fruits | Range*
blackberry, domestic - low
blackberry, wild - high
boysenberry - med.
choke cherry - high
wild crabapple - high
market cranberry - low
Swedish (lignon) cranberry - high
currant - med.
elderberry - med. to high
gooseberry - med.
huckleberry - med.
loganberry - med.
mulberry - med.
quince - med.
raspberry - med.
Seeds | Range*
apple seeds - high
apricot seed - high
buckwheat - med.
cherry seed - high
flax - med.
millet - med.
nectarine seed - high
peach seed - high
pear seeds - high
plum seed - high
prune seed - high
squash seeds - med.
Beans | Range*
black - low
black-eyed peas - low
fava - high
garbanzo - low to med.
green pea - low
kidney - low to med.
lentils - med.
lima, U.S. - low
lima, Burma - med.
mung - med. to high
shell - low
Nuts (all raw) | Range*
bitter almond - high
cashew - low
macadamia - med. to high
Sprouts | Range*
alfalfa - med.
bamboo - high
fava - med.
garbanzo - med.
mung - med.
Leaves | Range*
alfalfa - high
beet tops - low
eucalyptus - high
spinach - low
water cress - low
Tubers | Range*
cassava - high
sweet potato - low
yams - low
Range* 1000 mg ( milligrams ) = 1 gram
High — above 500 mg nitriloside per 100 grams of food.
Medium — above 100 mg nitriloside per 100 grams of food.
Low — below 100 mg nitriloside per 100 grams of food.
"Peter Moran" <pmoranATbordernet.com.au> wrote
Under the title B-17
The study ----
Long Term Follow-up of cancer patients using Contreras, Hoxsey and Gerson
therapies. Austin S et al. J. Naturopathic Med. 1994; 5(1):74-75
As of 12 January 1999, of 11 patients entered into the study, 9 (81%)
survived one year, 5 (45%) survived two years, and at this time, 4 have
survived three years. Two patients are alive and doing well: one at three
years and the other at four years. These results are far above the 25%
survival at one year and 10% survival at two years for all stages of
pancreatic adenocarcinoma reported in the National Cancer Data Base from
1995. This pilot study suggests that an aggressive nutritional therapy with
large doses of pancreatic enzymes led to significantly increased survival
over what would normally be expected for patients with inoperable pancreatic
The pancreatic cancer trial by Nicholas J. Gonzalez, MD, a physician in
private practice in New York, will test whether use of pancreatic enzymes
from pigs, combined with dietary supplements, nutritional therapy, and
coffee enemas, is effective in treating pancreatic cancer.
Dr. Gonzalez' therapeutic ideas have a long history, dating back to
anecdotal reports in medical journals in the early decades of the 20th
More significantly, he drew on the work of William Kelley, DDS, a dentist
whom Dr. Gonzalez termed "eccentric and controversial" but who treated more
than 10,000 patients by nutritional means and kept detailed records.
At this point, Dr. Good and Dr. Gonzalez realized that even though Kelley's
results were extraordinary, in fact unparalleled in medicine, they were of
little use in the treatment of cancer unless they could be independently
reproduced. This is the stage of the research at present. For the last 3
years, Dr. Gonzalez has been using Kelley's protocol with a few
modifications of his own in the treatment of terminal cancer.
The Research Division has been evaluating Dr. Gonzalez' results over the
last four months, including numerous site visits. We have interviewed
patients at length by phone, met with a number of patients and reviewed case
histories of some 40 terminal cancer patients. The results are indeed
We have seen excellent outcomes with a pancreatic cancer case (metastatic to
the liver) diagnosed at Mayo Clinic. We have seen return to work for over
two years in a case of metastatic melanoma previously operated on twice
(unsuccessfully) at Memorial Sloan-Kettering. We have seen long-term
survival in terminal lung cancers. We also have two MBL [Mutual Benefit
Life] insureds under Dr. Gonzalez' care, one of whom has come back from
terminal ovarian cancer (two years ago) to the point where she is preparing
to return to work (this is an LTD claim). The other refused a bone marrow
transplant after experiencing recurrence of Hodgkin's disease following
three different regimens of chemotherapy and radiation that produced
short-term remissions. She has been on Dr. Gonzalez' protocol for 15
The Hoxsey Therapy is the oldest continuously used unconventional or
alternative nontoxic cancer therapy in North America. Ironically, from the
1920s until recently, it was usually vilified as the leading example of
medical quackery in modern times. Among the Hoxsey Therapy's other
a.. It was the first North American alternative cancer therapy to set up a
base in Mexico
b.. It is the leading example of herbal or botanical medicine for cancer
c.. It has a remarkable and unique cultural and political history
d.. Even more remarkable are the reports about its successful clinical
history in the face of daunting opposition and odds
e.. It represents a leading example of an uncompromising, primary therapy
f.. It was decades ahead of its time in many ways - particularly in its
underlying theory that cancer is a systemic condition that might be
successfully treated by natural chemotherapy, in this case of a nontoxic
g.. It has made contributions to the empirical use - and our understanding
of the importance - of group therapy, attitudinal healing, and nutritional
Mexico is pretty good on dentistry and its closer.
I had an aunt who used to go there for laetrile.
The medical establishment wrote her off because she had breast cancer and her insurance ran out. Gave her 6 months. She went to Mexico for laetrile and lived 10 years more. Got hit by a beer truck in downtown Tijuana.
-435 for a post count, no obvious advertisements, no percieved motive other than to help people with a personal testimony!
Gentlemen, eat your chili peppers. Habanero, jalapeno, Scotch bonnet -- those hot but tasty varieties of the capsicum frutescens have multiple health benefits -- including the ability to drive prostate cancer cells to kill themselves, researchers announced yesterday.
According to a team from the University of California at Los Angeles and Cedars-Sinai Medical Center, the hot stuff in peppers -- capsaicin -- caused 80 percent of active prostate cancer cells growing in mice to "follow the molecular pathways leading to apoptosis," or cell death.
The cancer cells literally committed suicide. What's more, the cancer tumors of the mice treated with a hot pepper extract were one-fifth the size of untreated mice.
"Capsaicin had a profound anti-proliferative effect on human prostate cancer cells in culture," said Dr. Soren Lehmann. "It also dramatically slowed the development of prostate tumors formed by those human cell lines grown in mouse models."
What does this mean in the kitchen? Tex-Mex or curry fans are in luck: the hotter the pepper, the more the benefit. According to Dr. Lehmann, the mice were fed a dose of pepper extract equivalent to what a normal man might consume -- 400 milligrams of extract three times a week. That amount translates to three to eight fresh habanero peppers.
Medically speaking, capsaicin inhibited the action of NF-kappa Beta, a substance found in cells that causes them to grow out of control. Capsaicin also regulates certain proteins that effect the growth of the cells.
"Increased concentrations of capsaicin caused more prostate cancer cells to freeze in a non-proliferative state," according to the study.
American men develop prostate cancer more than any other type of malignancy -- 232,000 new cases are diagnosed in the United States annually; 30,000 men die of prostate cancer in the United States each year.
Hot peppers have received accolades from researchers in recent years for their antioxidant, or cancer-fighting, effects. Anti-inflammatory properties in peppers have been tapped for treatment of migraines, arthritis and muscle pain. Hot peppers also have been found to suppress appetites and clear a stuffy head; they can aggravate existing heartburn but not cause it.
They are a good source of vitamins A, C and E, folic acid and potassium. Peppers are low in calories and sodium and contain no carbohydrates. Their taste has spawned numerous appreciation societies around the world, not to mention global competitions to determine the hottest variety on the planet.
Still, the chili pepper came under fire in a 2002 Yale University report that established a link between the hot pods and stomach cancer in Mexican workers who ate from 9 to 25 jalapenos a day. The claim has been disputed by other researchers who found that rates of stomach cancer declined in the United States -- though consumption of salsa, chili and other hot foods actually had increased.
Last edited by madthumbs on Fri Mar 17, 2006 5:35 am; edited 1 time in total
Thu Mar 16, 2006 7:30 pm
Joined: 22 Feb 2006 Posts: 8599 Location: Fingerlakes - NY usa
Red chilli peppers contain cancer-busting chemicals: study
ISLAMABAD: Researchers are rolling out the spice rack against cancer with studies showing that ginger and the hot element in red chili peppers could kill tumour cells.
The ginger and chilli-pepper studies were presented during the annual meeting of the American Association for Cancer Research in Washington. Researchers at the University of Pittsburgh experimented on mice toshow that capsaicin, the active ‘hot’ ingredient in the pepper, killed pancreatic cancer cells through the body’s normal process for clearing defective cells.
“We discovered that capsaicin fed orally to mice with human pancreatic tumours was an extremely effective inhibitor of the cancer process, inducing apoptosis (natural cell death) in cancer cells,” said an assistant professor of pharmacology at Pitt and lead author of the study Sanjay Srivastava.
Tumours treated with capsaicin were half the size of tumours found in mice that were treated with saline solution by the end of the study. Scientists at the University of Michigan Comprehensive Cancer Centre tested powdered ginger against ovarian-cancer-cell cultures in a lab and planned to work with mouse in the next phase.
The research found that ginger caused cell death in all the ovarian-cancer cell lines tested. Moreover, the spice caused cancer cells to be destroyed both through the normal cell-death process and through another mechanism that involves the cells digesting themselves.
The second mechanism is important, because it might offer a way around the difficult tendency of ovarian cancer cells to become resistant to conventional chemotherapy, said an assistant professor of obstetrics and gynaecology at the UM Medical School and author of the study Dr Rebecca Liu.
Ginger is known to be effective at controlling inflammation, which contributes to the development of ovarian cancer cells. By halting the inflammatory reaction, the researchers suggest, ginger also stops cancer cells from growing.
“We found that ginger induced cell death at a similar or better rate than platinum-based chemotherapy drugs used to treat ovarian cancer,” said an oncology fellow and co-author of the study Dr Jennifer Rhode.
Another review of studies evaluating the use of soy supplements against breast cancer finds that there is only a slight benefit. A study published in the Journal of the National Cancer Institute says that while soy intake may be associated with a small reduction in the risk of breast cancer, the evidence is too weak to recommend widespread use of the supplements.
The soy connection has been widely debated and tested for nearly three decades since scientists first noted that Asian women have much lower breast-cancer rates than women in Western nations. The scientists have also observed an increase in breast-cancer rates among the Asian women who moved to the United States.
Scientists at Johns Hopkins University and Georgetown University pooled the results of 18 studies published between 1978 and 2004 that looked at the association between soy intake and breast-cancer risk.
The researchers found an overall relative reduction in breast-cancer risk for soy-eaters — a modest 14 percent for Caucasian women — but the effect was not statistically significant for Asian women.
“We found that soy-food intake was associated with a reduced breast-cancer risk, but the data is not adequate to provide a clear answer to recommend soy foods to prevent breast cancer,” said a professor of oncology at Georgetown’s Lombardi Cancer Centre Leena Hilakivi-Clarke.
The researchers warned women against taking high-dose soy supplements. APP
WEDNESDAY, April 5 (HealthDay News) -- Ordinary ginger causes ovarian cancer cells to die, highlighting the spice's potential in fighting the killer disease, a new study found.
Not only did ginger trigger ovarian cancer cell death, it did so in a way that may prevent tumor cells from becoming resistant to treatment, a common problem with chemotherapy.
The preliminary findings from researchers at the University of Michigan Comprehensive Cancer Center were presented Tuesday at the annual meeting of the American Association for Cancer Research in Washington, D.C.
The Michigan team dissolved ginger powder in a solution and applied it to ovarian cancer cells in a laboratory. Ginger caused two kinds of cancer cell death. The first is called apoptosis, in which the cells essentially commit suicide. The second type of cell death is autophagy, in which cells digest or attack themselves.
"Most ovarian cancer patients develop recurrent disease that eventually becomes resistant to standard chemotherapy -- which is associated with resistance to apoptosis. If ginger can cause autophagic cell death in addition to apoptosis, it may circumvent resistance to conventional chemotherapy," study author Dr. J. Rebecca Liu, assistant professor of obstetrics and gynecology, explained in a prepared statement.
The researchers' next step is to determine if ginger can achieve similar results in animals.
It's already known that ginger helps control inflammation, which contributes to the development of ovarian cancer cells. By halting inflammation, ginger may stop cancer cells from growing, the researchers suggested.
Tue Apr 11, 2006 4:32 pm
Joined: 22 Feb 2006 Posts: 8599 Location: Fingerlakes - NY usa
Prostate tumours shrunk by lycopene, vitamin E combo
14/04/2006 - A combination of lycopene and vitamin E suppressed the growth of prostate cancer in mice, but had no effect when used independently, say Dutch researchers.
The role of tomato and its extracts to protect against prostate cancer has been reported in several epidemiological studies, which lycopene supplement producers have been quick to promote. The new research, led by Professor Wytske van Weerden from Erasmus MC in Rotterdam, casts doubt on the effectiveness of lycopene by itself, and suggests a synergetic effect with vitamin E, another nutrient naturally found in tomatoes.
The study, published in the May issue of the Journal of Nutrition (Vol. 136, pp. 1287-1293), followed the effects on differing supplementation doses and combinations of lycopene and vitamin E on 54 mice inoculated with prostate cancer cells.
The mice were divided into six equal groups. The mice were fed a standard low vitamin E rodent diet, and three days after inoculation the diet was supplemented with one of the following: nothing (placebo); lycopene only (5 milligrams per kilogram of body weight (mg/kg BW) or 50 mg/kg BW); vitamin E only (5 mg/kg BW or 50 mg/kg BW); or vitamin E plus lycopene (5 mg/kg BW each).
BASF provided both nutrients: lycopene was provided as LycoVit 10 per cent, and vitamin E as alpha-tocopherol 50 per cent powder.
The researchers found that, after 95 days of supplementation, none of the single supplements, regardless of dose, had any effect on the tumour size.
“Compared with the control, the combined mixture of lycopene and vitamin E, at five mg/kg BW each, suppressed the growth of the prostate xenograft by 73 per cent at day 42,” wrote lead author Jacqueline Limpens.
Consequently, the mice receiving the combined supplement also lived 40 per cent longer than the placebo group.
While the single supplements did not effect tumour size, the group receiving five mg/kg BW of lycopene did show slower tumour growth and a 19 per cent longer survival rate.
The researchers also reported that levels of the so-called prostate specific antigen (PSA), a protein that is used as a marker for the disease, tended to be lower in the combo supplement group. Plasma PSA levels were proportional to tumour size for all groups, adding support for the use of PSA as a marker of the disease.
Previous research has linked the benefits of tomatoes to the lycopene content of the fruit, conclusions that are not in-line with the results of Limpens and her colleagues.
Indeed, it appears as if lycopene and vitamin E exert a cooperative interaction to protect against prostate cancer growth. The mechanism, say the researchers, remains speculative but could be due to lower oxidative stress, altered hormone and growth factor signals, or apoptosis (programmed cell death).
“On the basis of our findings… we are currently assessing the effects of lycopene-vitamin E supplementation on rising PSA-levels in an exploratory phase II clinical prostate cancer trial,” said the researchers.
According to the European School of Oncology, over half a million news cases of prostate cancer are diagnosed every year world wide, and the cancer is the direct cause of over 200,000 deaths. More worryingly, the incidence of the disease is increasing with a rise of 1.7 per cent over 15 years.
Mon Apr 17, 2006 2:24 pm
Joined: 22 Feb 2006 Posts: 8599 Location: Fingerlakes - NY usa
So many are frustrated. It is becoming clear, in regards to health information, that pharmaceutical manufacturers hold a grip over the public by controlling government agencies and the news media. An example is the recent report that intravenous vitamin C may cure cancer. That report was relegated to an obscure medical journal and went unreported by major news outlets (New York Times, USA Today, CNN, FoxNews, ABC, NBC, CBS, WebMD) which better than 75% of the public rely upon for up to date health information.
Wed Apr 19, 2006 11:42 am
Joined: 22 Feb 2006 Posts: 8599 Location: Fingerlakes - NY usa
Sunshine heals cancer, and the FDA is powerless to stop it,
Do you realize that one of the most powerful cures for cancer is streaming over our heads each and every day, free of charge? It's sunlight, which is astounding in its ability to prevent and cure cancer. If it were a mainstream drug, it would probably make the cover of Time magazine and be heralded as the greatest medical breakthrough in the history of modern science. It's that good.
Sunlight exposure reduces the risk of many cancers by more than 50 percent and even helps reverse certain types of cancers through the creation of vitamin D in the body. It's a magnificent natural healing modality, and it's been right in front of our eyes, every single day, since before Homo sapiens even evolved on this planet. Yet somehow, after spending billions of dollars on so-called medical research to find "cures" for various cancers, almost no one from the world of mainstream medicine has yet acknowledged the healing power of natural sunlight and vitamin D.
None of them have actually prescribed sunlight to patients, except for perhaps a handful of pioneering researchers like Dr. Michael Holick, who was attacked for speaking out about the truth of sunlight and cancer. By and large, the medical community has not only ignored this truly miraculous cure for many types of cancer; it has worked hard to discredit it.
If there were ever a reason to lose faith in conventional medicine, or so-called modern medicine, it is simply the fact that one of the greatest cures and prevention strategies for cancer goes completely ignored by nearly the entire conventional medical community. It's as if there were a miracle medicine invented, but conventional medical doctors didn't want anyone to find out about it. Why aren't researchers promoting cures that are available for free?
Cells, whether cancerous or normal can only live and reproduce (undergo mitosis) in a Ph range of between 6.5 and 7.5. A healthy cell has a Ph of 7.35 while a cancer cell is more acidic. Cesium when taken orally will raise the Ph of cancer cells, but not that of normal cells. When the Ph of a cancer cell goes above 7.5 it dies and if it goes above 8.0 it will die in a matter of hours.
What can enter a cancer cell
Every cell in the body is like a little battery. To successfully bring nourishment in, and take poisons out, it has to be fully charged. In a cancerous cell, the charge (called cell voltage) drops from 90 millivolts to less than 40 millivolts. When the cell voltage gets to the very bottom, only 5 substances can pass in or out of the cell. They are water, sugar, potassium, cesium and rubidium. Oxygen cannot enter into a cancer cell. So you see, even if there is a lot of oxygen in the blood, it won't get into the cell. Cesium, because of its electrical properties can still enter the cancerous cell. When it does so, because of it's extreme alkalinity, the cell dies. Luckily, healthy cells are not affected by cesium because their cell voltage allows them to balance themselves. The only side effect is a loss of potassium which can be remedied with eating a few bananas and potatoes.
It is interesting to note that cancer is virtually unknown among the Hopi Indians of Arizona and the Hunza of Northern Pakistan, so long as they stay in the same environment. This strongly suggests that something they are consuming is protecting them from cancer. The Hopi water is rich in Rubidium and potassium. The Hunza water is rich in Cesium and potassium, making both of the water supplies rich with very caustically (alkaline) active metals.
In his publication, Cesium therapy in cancer patients, Dr. Sartori describes the 2 week treatment of 50 last stage, metastasized, terminal cancer patients (13 comatose), with Cesium salts. All were expected to die within weeks, with the survival rate being less than one in ten million. After 2 weeks, 13 died with autopsies showing no presence of cancer. After 12 months, 12 more had died, but 25, an astounding 50% survived.
*Cesium has no natural radioactive form, and should not be confused with Cesium 137 which is artificially produced.
Cancer cells are very weak, far weaker than healthy cells. It is very easy to kill cancer cells if you can create the right environment. The following protocols are deadly to cancer cells, yet harmless if not outright beneficial to healthy cells.
The High pH Environment
Cancer cells live in an acidic environment, but perish in an alkaline, high Ph, environment. Although many diets can help you alkalinize your body, nothing works as fast as Cesium Carbonate or Cesium Chloride.
Cesium for Cancer
Cesium *, a crystalline salt has been used successfully for cancer for many years now. Cesium Chloride and Cesium Carbonate work by raising the cancer cell's Ph to a highly alkaline state. Although many anti-cancer diets also produce an alkaline state, they simply cannot do so as quickly or as fully as Cesium can.
Cesium Therapy in Cancer patients
Certain foods contain biologically active compounds and/or ingredients, i.e., vitamins, inorganic salts, organic compounds, essential fatty acids, minerals, and chelating agents which may either precipitate or prevent cancer development. The relationship between dietary consumption and cancer development is not clear and further investigation continues. Noteworthy is the report on the presence of high levels of cesium [Cs] and rubidium [Rb] in food along with availability of various supportive compounds as vitamins A and C, along with zinc and selenium in diet of populations residing in areas with low incidence of cancer e.g., the Hopi Indian territory in Arizona, the Hunza area in North Pakistan, and the volcanic regions of Brazil. The diet of these populations is similar to the nutritive requirements for the high Ph cancer therapy developed by Brewer's subsequent series of physical experiments with cancer cells. In these tests the presence of Cs+ or Rb+ in the adjacent fluids of the tumor cell is believed to raise the Ph of the cancer cell where mitosis will cease resulting in reduction of life span of the cancer cell. The introduction of such alkaline Ph by these alkali salts may also neutralize the acidic and toxic material within the cancer cell. This report combines the use of CsCl with various supportive agents. which have been hypothesized both to enhance the entry of Cs+ into the cancer cell and to stimulate the immune response, in the treatment of various cancers.
Treatment was performed on 50 patients during the last three years at Life Sciences Universal Medical Clinics in Rockville MD and in Washington D.C. All patients were terminal subjects with generalized metastatic disease. Forty-seven of the 50 patients studies had received maximal modalities of treatment, i.e., surgery, radiation, and various chemotherapy, before metabolic Cs-treatment was initiated. Three patients were comatose and 14 of the patients were considered terminal due to previous treatments outcome and cancer complications. The type of cancer of the patients studied and their number is detailed in table 1.
The Cs-treatment was given in conjunction of other supportive compounds under diet control in addition to the utilization of specific compounds to produce adequate circulation and oxygenation. According to individual cases CsCl was given at daily dosages of 6 to 9 grams in 3 equally divided doses, with vitamin A-emulsion (100,000 to 300,000 U), vitamin C (4 to 30 grams), zinc (80 to 100 mg) selenium (600 to 1,200 mcg) and amygdalin (1,500 mg) in addition to other supplementations according to the specific needs of the patient. The diet consisted mainly of whole grains, vegetables, linolenic acid rich oils (linseed, walnut, soy, wheat germ) and other supplemental food. To increase efficiency of the treatment and improve the circulation and oxygenation, the patients received the chelating agent EDTA, dimethylsulfoxide (DMSO) and also a combination of vitamins, K and Mg salts.
Table 1 summarizes the results of the Cs-treatment of 50 cancer patients studied over 3 years. They had generalized metastatic disease, except for 3 patients. Initial death occurrences for the initial 2 week treatment was in the same order and magnitude of these recorded for the 12 month period. The percent of survival of breast, colon, prostate, pancreas, and lung cancer accounted to approximately, 50% recovery which was higher than that noted for liver cancer and the lymphoma patients treated. An overall 50% recovery from cancer by the Cs-therapy was determined in the 50 patients treated. Data from the autopsy made indicated the absence of tumors in patient dying within 14 days of the Cs-treatment. One of the most striking effects of the treatment was the disappearance of pain in all patients within 1 to 3 days after initiation of the Cs-therapy.
These studies were performed under my direction, initiated in April, 1981. Twenty-eight patients were initially treated with CsCl between April, 1981 to October, 1982. They were subjected to various cancer therapies, e.g., surgery, radiation, and chemotherapy, and were considered terminal cases with metastatic disease except for 3 patients who were not previously treated. Three patients were comatose at the time of the Cs treatment. Thirteen patients died within less than 2 weeks of treatment. Each patient showed a reduction in tumor mass by the Cs-treatment. Of the breast cancer patients, the most impressive effect was seen in a female patient who was comatose at the beginning of the Cs-treatment and was considered a terminal case. The Cs-therapy, with other ingredients used, was immediately instituted by nasogastric route because there was no cooperation from the patient. The daily CsCl dose given amounted to 30 grams, 10 grams given 3 times daily. The patient was able to leave after 5 days of treatment. However the patient's fall on the floor resulted in complications, i.e., fracture of the neck, and death. The autopsy revealed that the cancer metastasis had essentially eaten away her hip bone causing this tragic accident. The autopsy performed also showed the presence of very little cancer tissue.
The next most frequent cancer treated was of unknown primary. Treatment of 8 cases showed a death rate of 2 within 14 days of treatment and an additional 2 deaths within 12 months while 4 of the patients are still living. In one case, an autopsy was made in a patient after one week of Cs-treatment and showed a complete disappearance of the cancer. There were 7 cases of colon cancer patients who were treated with CsCl. Two of these patients died within 14 days, one of the patients had previous massive chemotherapy, and little time was available to restore her metabolic condition. The previous existing infiltration of the abdominal wall disappeared. However, no consent was given for an autopsy.
In one lymphoma case the patient displayed an unusually large abdomen which was hard and he weighed approximately 250 pounds. The massively enlarged abdomen began to decline in volume, i.e., a loss of approximately 120 pounds of body weight was noted after 3 months of the Cs- therapy. The spleen which was originally maximally enlarged and reaching into the pelvis was reduced to almost normal size. The liver position was down to about the level of the umbilicus and was also reduced to normal size in 3 months. The patient is still living after 3 years after his discharge. Unfortunately, there is no follow-p on this patient and he is being maintained on chemotherapy.
The results presented demonstrate the rate of efficacy of CsCl in cancer therapy. The total 50 cancer cases studied show an impressive 50% survival rate. This confirms the work of Messiha reported in these proceedings showing that the higher the dose it is, the more effective it seems to be. The autopsy obtained from the patient whose death was attributed to traumatic fracture of the neck, indicated that cancer had been initially further advanced resulting in bone destruction. However, the absence of cancer after the massive CsCl dose used in this case is demonstrable of the Cs-therapy. It appears that both dosage, i.e., as much as 30 grams/day and route of drug administration, i.e., nasogastric pathway, might have contributed to the patients rapid recovery. It should be noted, however, that CsCl dose regimens should not exceed 20 to 40 grams due to side effects, mainly nausea, and diarrhea. The authors personal experience with CsCl after an acute dose of 40 grams CsCl indicate that extensive nausea and parethesia around the mouth are the major side effects. This is probably due to K depletion. The usual dose used in the clinic ranges from 2 to 3 grams given by mouth 3 times daily. At a later time, at which time there is no indication of cancer presence, the CsCl dosage will be reduced to a preventative dose between .5 and 1 gram a day.
The lymphoma case presented shows that CsCl efficiently reduced massive enlargements of spleen and liver as well as maximal ascites, causing an abdominal configuration of a tight, hard hemisphere, to almost normalize after 3 months of therapy. This period of time was required to eliminate such a massive volume resulting in the reduction of the body weight noted.
The clinical efficacy of CsCl high pH metabolic therapy is best demonstrated by a recent case of primary liver cancer (not included in the 50 cases reported in this study). The patient was a 39 year old female teacher who was terminal. She was brought on a stretcher on April 25, 1984 with a large liver tumor extending approximately 3 cm below the umbilical level. The treatment was then immediately instituted. This consisted of administration of CsCl, Beta-carotene, Vitamin C, Zn, Se, Mn, Cr, and K salts by the oral route in addition to a concomitant massive IV doses of ascorbate, K, Mg, Zn, Cn, Mn, Cr salts, B complex vitamins, folic acid, DMSO and heparin. After 5 consecutive treatment regimens EDTA was introduced to the therapy and the minerals present in the solution were discontinued. On May 10, 1984, the patient was discharged, returned home walking without assistance and displaying a smile on her face. The liver tumor had shrunk to 5 cm above the umbilicus. The determination of alphafetoprotein (AFP), a specific marker for liver cancer, rare embronal cancer and teratomas, decreased from the unusually high value of 39,000 units, compared to normal levels of 13 units, measured before initiation of Cs-therapy, to 5000 units obtained on the last day of treatment.
The mechanism of action of Cs in cancer has been little studied. Both Cs+ and Rb+ can specifically enter the cancer cells and embryonic cells, but not normal adult cells has been demonstrated by Brewer. The cancer cells contain high amounts of hydrogen ions rendering them acidic and they also contain high Na+ levels than found in normal cells. If Cs+ or Rb+ can enter the cancer cells then the pH increases from as low as 5.5 to over pH 7.0. At a pH of 7.6 the cancer cell division will stop, at a pH of 8.0 to 8.5 the lifespan of it is considerably shortened (only hours). In one case, the author has observed the shrinkage of metastases of breast cancer after one hour of Cs-treatment. Two days later wrinkles of the skin appeared where the tumor was present. In another case of a colon cancer with massive metastasis, of massive infiltration of the abdominal wall, liver and other tissues, seemed to have been reduced within 24 hours and continuing rapidly until the demise of the patient on the 14th day of the Cs-treatment.
The uric acid levels measured at the onset of treatment was approximately 3.5 units which was increased to over 20 units, suggesting massive breakdowns of DNA, which produces the uric acid output. Therefore, destruction of nuclear acids, as reflected by a significant rise in the uric acid, may be used as a predictive measurement for treatment outcome. The failure of uric acid elevation may be indicative of lack of destruction of cancer cells. This has proven to be a very consistent finding in our clinic.
There are certain factors which may enhance the Cs-therapy. The Cs-penetration into the cancer cells can be increased by the following three methods: The first approach resides in broadening the electron donor capacity of the cancer cell membrane by the application of cyanide, an electron donor radical as found in nitriles (amygdalin, Laetrile, mandelonitrite, prunasin, ficin, cassivin), by selenium oxide, an electron donor radical, or by the use of DMSO. The second approach enhances the potential gradient across the cancer cell membrane by the utilization of weak acids like ascorbic acid (Vitamin C) and retinoic acid (Vitamin A). The third method attempts to improve the circulation to the tumor and facilitate the destruction of cross-linkages in the mucoid and fibrinous substances around the cancer cell. This can be achieved by chelation therapy, i.e., the use of EDTA as has been shown by Blumer who reported on the reduction of cancer incidence by 90% by chelating patients (an average of 15 chelations in 8 years). This approach also reduced cardiovascular disease by 50%. Other chelating agents can also be used. Moreover, the use of beta-carotene will lead to decomposition of blocking mucoid proteins mediated by electrical charges; Also, heparin, which acts through electrical charges, will inactivate the immune repelling and immune binding capacities of the blocking mucoid proteins. These approaches will hinder cancer growth and they are virtually atoxic.
It should be noted that certain behavioral characteristics "the cancer personality" of the cancer patient may interfere in any projected treatment modality. This has been reported by Lawrence LeShan in his book entitled "You can fight for your life." His studies suggested that cancer patients seeking treatment, e.g., chemotherapy, radiation or surgery, are probably motivated by a covert desire for death. For example, statements such as, "rather than undergoing any of those treatments, I would rather die in peace," or "I would never undergo any of those treatments or let anyone of my family undergo them because the effectiveness is unproven and the damage that is done with any of those treatments is higher than the effects." are often expressed. Thus, both chemotherapy and lifestyle changes may also contribute to an effective therapy.
The High Oxygen Environment
Nobel Laureate Otto Warburg demonstrated that normal cells would become irreversibly cancerous if the environment they rested in had their oxygen levels lowered by 35% for 48 hours.
Cancer Cells CANNOT Live in a High Oxygen Environment
A healthy individual has a blood oxygen level of between 98 and 100 as measured by a pulse oximeter.
Cancer patients routinely show very low oxygen levels in their blood, usually around 60.
According to Nobel prize laureate Dr. Otto Warburg, this low oxygen environment is one of the main reasons cancer cells form.
Unfortunately, the main traditional therapies for cancer, namely radiation and chemotherapy, also have been shown to drastically lower blood oxygen levels.
The High Enzyme Environment
Cancer cells develop a protein coating 13 times thicker than normal cells. This makes it difficult for the immune system to attack them. By ingesting high doses of pancreatin, you can actually dissolve cancer cells inside the body.
In the natural course of one's lifetime, millions of cancer cells develop, and are harmlessly digested by the immune system. The body uses pancreatin, a secretion from the pancreas to dissolve the cancer cells. As we age, the pancreas is less and less able to make this important substance. By taking pancreatin orally, it is possible to increase the levels of its active ingredients in the blood, thereby helping the body break down the cancer cells and remove them from circulation.
Pancreatin as a digestive enzyme is available from any health food store in the country, however this type of pancreatin is useless for the cancer patient. The active ingredients in pancreatin which have shown to have tumor dissolving abilities are trypsin and chymotrypsin. These ingredients were taken out of virtually all the pancreatin supplements available to consumers years ago. These active ingredients are being bought in massive quantities by the sewerage industries to digest the sewerage into less noxious forms.
This is exactly what is needed in the human body. Our own internal sewerage needs to be dissolved, and to do this, the body uses trypsin and chymotrypsin.
The high pH therapy for cancer tests on mice and humans.
1: Pharmacol Biochem Behav. 1984;21 Suppl 1:1-5.
Mass spectrographic and isotope studies have shown that potassium, rubidium, and especially cesium are most efficiently taken up by cancer cells. This uptake was enhanced by Vitamins A and C as well as salts of zinc and selenium. The quantity of cesium taken up was sufficient to raise the cell to the 8 pH range. Where cell mitosis ceases and the life of the cell is short. Tests on mice fed cesium and rubidium showed marked shrinkage in the tumor masses within 2 weeks. In addition, the mice showed none of the side effects of cancer. Tests have been carried out on over 30 humans. In each case the tumor masses disappeared. Also all pains and effects associated with cancer disappeared within 12 to 36 hr; the more chemotherapy and morphine the patient had taken, the longer the withdrawal period. Studies of the food intake in areas where the incidences of cancer are very low showed that it met the requirements for the high pH therapy.
Sun May 07, 2006 9:33 pm
Joined: 22 Feb 2006 Posts: 8599 Location: Fingerlakes - NY usa
PORTLAND, Ore. - For many men, a finding by Oregon researchers sounds too good to be true: an ingredient in beer seems to help prevent prostate cancer, at least in lab experiments. The trouble is you'd theoretically have to drink about 17 beers a day for any potential benefit. And no one's advising that.
Researchers at Oregon State University say that the compound xanthohumol, found in hops, inhibits a protein in the cells along the surface of the prostate gland. The protein acts like a switch that turns on a variety cancers, including prostate cancer.
Dr. Richard N. Atkins, CEO of the National Prostate Cancer Coalition, said the experiments are encouraging and "perhaps men could take it in pill form someday."
He noted an ingredient in tomatoes, lycopene, has previously been linked to prostate cancer prevention.
"It's every man's dream to hear that beer and pizza can prevent cancer," he said. "However, the 17 beers and four large pizzas needed to get enough xanthohumol and lycopene to help prevent prostate cancer is unfortunately not advised."
Atkins noted that drinking 17 beers a day can lead to alcoholism and cirrhosis of the liver, and overdoing it on pizza can lead to obesity and other health problems.
"Food, no matter how helpful it may be, is not a full preventive for prostate cancer," he said.
Tue Jun 13, 2006 11:32 am
Joined: 22 Feb 2006 Posts: 8599 Location: Fingerlakes - NY usa
THC destroyed incurable brain tumors?
By Raymond Cushing, AlterNet. Posted May 31, 2000.
In 1974 researchers learned that THC, the active chemical in marijuana, shrank or destroyed brain tumors in test mice. But the DEA quickly shut down the study and destroyed its results, which were never replicated -- until now.
The term medical marijuana took on dramatic new meaning in February, 2000 when researchers in Madrid announced they had destroyed incurable brain tumors in rats by injecting them with THC, the active ingredient in cannabis.
The Madrid study marks only the second time that THC has been administered to tumor-bearing animals; the first was a Virginia investigation 26 years ago. In both studies, the THC shrank or destroyed tumors in a majority of the test subjects.
Most Americans don't know anything about the Madrid discovery. Virtually no major U.S. newspapers carried the story, which ran only once on the AP and UPI news wires, on Feb. 29, 2000.
The ominous part is that this isn't the first time scientists have discovered that THC shrinks tumors. In 1974 researchers at the Medical College of Virginia, who had been funded by the National Institute of Health to find evidence that marijuana damages the immune system, found instead that THC slowed the growth of three kinds of cancer in mice -- lung and breast cancer, and a virus-induced leukemia.
The DEA quickly shut down the Virginia study and all further cannabis/tumor research, according to Jack Herer, who reports on the events in his book, "The Emperor Wears No Clothes." In 1976 President Gerald Ford put an end to all public cannabis research and granted exclusive research rights to major pharmaceutical companies, who set out -- unsuccessfully -- to develop synthetic forms of THC that would deliver all the medical benefits without the "high."
The Madrid researchers reported in the March issue of "Nature Medicine" that they injected the brains of 45 rats with cancer cells, producing tumors whose presence they confirmed through magnetic resonance imaging (MRI). On the 12th day they injected 15 of the rats with THC and 15 with Win-55,212-2 a synthetic compound similar to THC. "All the rats left untreated uniformly died 12-18 days after glioma (brain cancer) cell inoculation ... Cannabinoid (THC)-treated rats survived significantly longer than control rats. THC administration was ineffective in three rats, which died by days 16-18. Nine of the THC-treated rats surpassed the time of death of untreated rats, and survived up to 19-35 days. Moreover, the tumor was completely eradicated in three of the treated rats." The rats treated with Win-55,212-2 showed similar results.
The Spanish researchers, led by Dr. Manuel Guzman of Complutense University, also irrigated healthy rats' brains with large doses of THC for seven days, to test for harmful biochemical or neurological effects. They found none.
"Careful MRI analysis of all those tumor-free rats showed no sign of damage related to necrosis, edema, infection or trauma ... We also examined other potential side effects of cannabinoid administration. In both tumor-free and tumor-bearing rats, cannabinoid administration induced no substantial change in behavioral parameters such as motor coordination or physical activity. Food and water intake as well as body weight gain were unaffected during and after cannabinoid delivery. Likewise, the general hematological profiles of cannabinoid-treated rats were normal. Thus, neither biochemical parameters nor markers of tissue damage changed substantially during the 7-day delivery period or for at least 2 months after cannabinoid treatment ended."
Guzman's investigation is the only time since the 1974 Virginia study that THC has been administered to live tumor-bearing animals. (The Spanish researchers cite a 1998 study in which cannabinoids inhibited breast cancer cell proliferation, but that was a "petri dish" experiment that didn't involve live subjects.)
In an email interview for this story, the Madrid researcher said he had heard of the Virginia study, but had never been able to locate literature on it. Hence, the Nature Medicine article characterizes the new study as the first on tumor-laden animals and doesn't cite the 1974 Virginia investigation.
"I am aware of the existence of that research. In fact I have attempted many times to obtain the journal article on the original investigation by these people, but it has proven impossible." Guzman said.
In 1983 the Reagan/Bush Administration tried to persuade American universities and researchers to destroy all 1966-76 cannabis research work, including compendiums in libraries, reports Jack Herer, who states, "We know that large amounts of information have since disappeared."
Guzman provided the title of the work -- "Antineoplastic activity of cannabinoids," an article in a 1975 Journal of the National Cancer Institute -- and this writer obtained a copy at the University of California medical school library in Davis and faxed it to Madrid.
The summary of the Virginia study begins, "Lewis lung adenocarcinoma growth was retarded by the oral administration of tetrahydrocannabinol (THC) and cannabinol (CBN)" -- two types of cannabinoids, a family of active components in marijuana. "Mice treated for 20 consecutive days with THC and CBN had reduced primary tumor size."
The 1975 journal article doesn't mention breast cancer tumors, which featured in the only newspaper story ever to appear about the 1974 study -- in the Local section of the Washington Post on August 18, 1974. Under the headline, "Cancer Curb Is Studied," it read in part:
"The active chemical agent in marijuana curbs the growth of three kinds of cancer in mice and may also suppress the immunity reaction that causes rejection of organ transplants, a Medical College of Virginia team has discovered." The researchers "found that THC slowed the growth of lung cancers, breast cancers and a virus-induced leukemia in laboratory mice, and prolonged their lives by as much as 36 percent."
Guzman, writing from Madrid, was eloquent in his response after this writer faxed him the clipping from the Washington Post of a quarter century ago. In translation, he wrote:
"It is extremely interesting to me, the hope that the project seemed to awaken at that moment, and the sad evolution of events during the years following the discovery, until now we once again draw back the veil over the anti-tumoral power of THC, twenty-five years later. Unfortunately, the world bumps along between such moments of hope and long periods of intellectual castration."
News coverage of the Madrid discovery has been virtually nonexistent in this country. The news broke quietly on Feb. 29, 2000 with a story that ran once on the UPI wire about the Nature Medicine article. This writer stumbled on it through a link that appeared briefly on the Drudge Report web page. The New York Times, Washington Post and Los Angeles Times all ignored the story, even though its newsworthiness is indisputable: a benign substance occurring in nature destroys deadly brain tumors.
Raymond Cushing is a journalist, musician and filmmaker. This article was named by Project Censored as a "Top Censored Story of 2000."
A new study published in Nature Reviews-Cancer provides an historic and detailed explanation about how THC and natural cannabinoids counteract cancer, but preserve normal cells.
The study by Manuel Guzmán of Madrid Spain found that cannabinoids, the active components of marijuana, inhibit tumor growth in laboratory animals. They do so by modulating key cell-signalling pathways, thereby inducing direct growth arrest and death of tumor cells, as well as by inhibiting the growth of blood vessels that supply the tumor.
The Guzman study is very important according to Dr. Ethan Russo , a neurologist and world authority on medical cannabis: "Cancer occurs because cells become immortalized; they fail to heed normal signals to turn off growth. A normal function of remodelling in the body requires that cells die on cue. This is called apoptosis, or programmed cell death. That process fails to work in tumors. THC promotes its reappearance so that gliomas, leukemias, melanomas and other cell types will in fact heed the signals, stop dividing, and die."
"But, that is not all," explains Dr. Russo: "The other way that tumors grow is by ensuring that they are nourished: they send out signals to promote angiogenesis, the growth of new blood vessels. Cannabinoids turn off these signals as well. It is truly incredible, and elegant."
In other words, this article explains several ways in which cannabinoids might be used to fight cancer, and, as the article says, "Cannabinoids are usually well tolerated, and do not produce the generalized toxic effects of conventional chemotherapies.
Usually, any story that even suggests the possibility of a new treatment for cancer is greeted with headlines about a "cancer cure" - however remote in the future and improbable in fact it might be. But if marijuana is involved, don't expect any coverage from mainstream media, especially since mainstream editors have been quietly killing this story for the past thirty years.
That's right, news about the abilility of pot to shrink tumors first surfaced, way back in 1974. Researchers at the Medical College of Virginia, who had been funded by the National Institutes of Health to find evidence that marijuana damages the immune system, found instead that THC slowed the growth of three kinds of cancer in mice -- lung and breast cancer, and a virus-induced leukemia.
The Washington Post reported on the 1974 study -- in the "Local" section -- on Aug. 18, 1974. Under the headline, "Cancer Curb Is Studied," it read in part: "The active chemical agent in marijuana curbs the growth of three kinds of cancer in mice and may also suppress the immunity reaction that causes rejection of organ transplants, a Medical College of Virginia team has discovered." The researchers "found that THC slowed the growth of lung cancers, breast cancers, and a virus-induced leukemia in laboratory mice, and prolonged their lives by as much as 36 percent."
"News coverage of the Madrid discovery has been virtually nonexistent in this country. The news broke quietly on Feb. 29, 2000 with a story that ran once on the UPI wire about the Nature Medicine article," complained MarijuanaNews.com editor Richard Cowan , who said he was only able to find the article through a link that appeared briefly on the Drudge Report Web page. "The New York Times, The Washington Post, and Los Angeles Times all ignored the story, even though its newsworthiness is indisputable: a benign substance occurring in nature destroys deadly brain tumors," added Cowan.
On March 29, 2001, the San Antonio Current printed a carefully researched, bombshell of a story by Raymond Cushing titled, "POT SHRINKS TUMORS; GOVERNMENT KNEW IN '74." Media coverage since then has been nonexistant, except for a copy of the story on Alternet .
It is hard to believe that the knowledge that cannabis can be used to fight cancer has been suppressed for almost thirty years , yet it seems likely that it will continue to be suppressed. Why?
According to Cowan, the answer is because it is a threat to cannabis prohibition . "If this article and its predecessors from 2000 and 1974 were the only evidence of the suppression of medical cannabis, then one might perhaps be able to rationalize it in some herniated way. However, there really is massive proof that the suppression of medical cannabis represents the greatest failure of the institutions of a free society, medicine, journalism, science, and our fundamental values," Cowan notes.
Millions of people have died horrible deaths and in many cases, familes exhausted their savings on dangerous, toxic and expensive drugs. Now we are just beginning to realize that while marijuana has never killed anyone, marijuana prohibition has killed millions.
Fri Jul 07, 2006 4:44 pm
Joined: 30 Jul 2006 Posts: 40 Location: upstate NY
I personaly liked macrobiotics. I was only 8 or 9 when we started it but it was a big change. Now that I think about it but feel I had better energy in general. We had initially gone to macrobiotic becouse my mom was diagnosed with ovarian cancer. She has never been one to go to the doctors, and has alwaylooked into what she could do naturally first. She did beat the cancer, and when she finally went to to the doctors in Houston, they said she must have seeked treatmen in Mexico (a lot cheeper) . I still think with diseases such as cancer, it is best to research all forms of treatment, and don't avoid learning all you can about the variuos types of treatments availible.
After reading all the pot/killing cancer cells stuff...maby it was a a little bet of both that heriped her. Eating a diet that consist of brown rice , bean and sea vegetables, and pickled plum paste along will a little pot smoking, may be what actully helped my mom. I did however read years ago about brownrices' ability to fight cancer cells.
Shes alive and doing awsome still today.